cell markers Search Results


anti ema 1  (Developmental Studies Hybridoma Bank)
94
Developmental Studies Hybridoma Bank anti ema 1
Anti Ema 1, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse multipotent mesenchymal stromal cell marker antibody panel  (R&D Systems)
94
R&D Systems mouse multipotent mesenchymal stromal cell marker antibody panel
Mouse Multipotent Mesenchymal Stromal Cell Marker Antibody Panel, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
mouse multipotent mesenchymal stromal cell marker antibody panel - by Bioz Stars, 2026-04
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human pluripotent stem cell antibody array  (R&D Systems Hematology)
94
R&D Systems Hematology human pluripotent stem cell antibody array
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Human Pluripotent Stem Cell Antibody Array, supplied by R&D Systems Hematology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
human pluripotent stem cell antibody array - by Bioz Stars, 2026-04
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pluripotent stem cell marker antibody panel plus  (R&D Systems)
93
R&D Systems pluripotent stem cell marker antibody panel plus
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Pluripotent Stem Cell Marker Antibody Panel Plus, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
pluripotent stem cell marker antibody panel plus - by Bioz Stars, 2026-04
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human mesenchymal stem cell marker antibody panel  (R&D Systems)
94
R&D Systems human mesenchymal stem cell marker antibody panel
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Human Mesenchymal Stem Cell Marker Antibody Panel, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
human mesenchymal stem cell marker antibody panel - by Bioz Stars, 2026-04
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macrophage inhibitory cytokine 1 mic1 1c3  (Novus Biologicals)
94
Novus Biologicals macrophage inhibitory cytokine 1 mic1 1c3
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Macrophage Inhibitory Cytokine 1 Mic1 1c3, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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human mouse rat neural progenitor cell marker antibody panel  (R&D Systems)
93
R&D Systems human mouse rat neural progenitor cell marker antibody panel
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Human Mouse Rat Neural Progenitor Cell Marker Antibody Panel, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
human mouse rat neural progenitor cell marker antibody panel - by Bioz Stars, 2026-04
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millipore mic1  (Novus Biologicals)
90
Novus Biologicals millipore mic1
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Millipore Mic1, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
millipore mic1 - by Bioz Stars, 2026-04
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spp1 recombinant protein  (MedChemExpress)
93
MedChemExpress spp1 recombinant protein
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Spp1 Recombinant Protein, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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dendritic cell dc  (Novus Biologicals)
90
Novus Biologicals dendritic cell dc
Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. <t>Pluripotent</t> stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells
Dendritic Cell Dc, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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epithelial cell marker pe keratin 18 ck18 mab  (Novus Biologicals)
91
Novus Biologicals epithelial cell marker pe keratin 18 ck18 mab
The distribution of PEDV in nasal cavity after intranasal inoculation in neonatal piglets. A For FACS analyses, neonatal piglets were nasally administered PEDV at indicated times. Then, individual cells isolated from the nasal mucosa were gated based on cytokeratin 18, and viral infection was detected by PEDV N protein staining, n = 3 from 3 piglets per group. B IHC results showed PEDV distribution in five cross-sections (I, II, III, IV and V) of the nasal cavity at 12 h post intranasal infection. The scale bar represents 20 μm. C Quantitative analysis of PEDV positive cells in the nasal cavity . All data shown are the mean results ± SD from three independent experiments. Statistical significance was tested using one-way ANOVA. ** P < 0.01.
Epithelial Cell Marker Pe Keratin 18 Ck18 Mab, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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neural stem cell markers nestin  (R&D Systems)
86
R&D Systems neural stem cell markers nestin
The distribution of PEDV in nasal cavity after intranasal inoculation in neonatal piglets. A For FACS analyses, neonatal piglets were nasally administered PEDV at indicated times. Then, individual cells isolated from the nasal mucosa were gated based on cytokeratin 18, and viral infection was detected by PEDV N protein staining, n = 3 from 3 piglets per group. B IHC results showed PEDV distribution in five cross-sections (I, II, III, IV and V) of the nasal cavity at 12 h post intranasal infection. The scale bar represents 20 μm. C Quantitative analysis of PEDV positive cells in the nasal cavity . All data shown are the mean results ± SD from three independent experiments. Statistical significance was tested using one-way ANOVA. ** P < 0.01.
Neural Stem Cell Markers Nestin, supplied by R&D Systems, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. Pluripotent stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells

Journal: BMC Cancer

Article Title: Tyrosine kinase inhibitor SU11274 increased tumorigenicity and enriched for melanoma-initiating cells by bioenergetic modulation

doi: 10.1186/s12885-016-2341-y

Figure Lengend Snippet: Bioenergetic modulation counteracts the protumorigenic action of the SU11274. a – b Protein lysates were prepared from untreated controls and Rel3 cells treated with 1 μM SU11274 in spheroid conditions for 7 days. Pluripotent stem cell array has shown expression of all pluripotency markers in Rel3 cells expanded as spheroids. SU11274 treatment further increased level of proteins associated with pluripotency in SU11274-treated cells correlating with their higher tumor initiating properties. Phosphokinase assay screening has shown several active intracellular signaling cascades. Phosphorylated forms of p53 (S392 and S46) were detected in the SU11274-treated cells (lower panel B), which correlates with SU11274 mediated inhibition of cell proliferation. RSK1/2/3 (S380) phosphorylation was increased in SU11274-treated cells. We detected phosphorylation of the following target kinases: ERK1/2 (T202/Y204, T185/Y187), P-RAS40 (T246), Akt 1/2/3 (S473), p38 alpha (T180/Y182), AMPK alpha1 (T183), CREB (S133), GSK-3 alpha/beta(S21/S9), WNK-1 (T60) and HSP60 in both treated and untreated cells

Article Snippet: Proteome profile of the melanosphere cells cultured for 7 days in the presence of 1 μM SU11274 was evaluated by the Proteome ProfilerTM Human Phospho-Kinase Antibody Array and the Human Pluripotent Stem Cell Antibody Array (R&D SystemsTM Inc., Minneapolis MN).

Techniques: Expressing, Inhibition, Phospho-proteomics

The distribution of PEDV in nasal cavity after intranasal inoculation in neonatal piglets. A For FACS analyses, neonatal piglets were nasally administered PEDV at indicated times. Then, individual cells isolated from the nasal mucosa were gated based on cytokeratin 18, and viral infection was detected by PEDV N protein staining, n = 3 from 3 piglets per group. B IHC results showed PEDV distribution in five cross-sections (I, II, III, IV and V) of the nasal cavity at 12 h post intranasal infection. The scale bar represents 20 μm. C Quantitative analysis of PEDV positive cells in the nasal cavity . All data shown are the mean results ± SD from three independent experiments. Statistical significance was tested using one-way ANOVA. ** P < 0.01.

Journal: Veterinary Research

Article Title: PEDV infection in neonatal piglets through the nasal cavity is mediated by subepithelial CD3 + T cells

doi: 10.1186/s13567-020-00883-w

Figure Lengend Snippet: The distribution of PEDV in nasal cavity after intranasal inoculation in neonatal piglets. A For FACS analyses, neonatal piglets were nasally administered PEDV at indicated times. Then, individual cells isolated from the nasal mucosa were gated based on cytokeratin 18, and viral infection was detected by PEDV N protein staining, n = 3 from 3 piglets per group. B IHC results showed PEDV distribution in five cross-sections (I, II, III, IV and V) of the nasal cavity at 12 h post intranasal infection. The scale bar represents 20 μm. C Quantitative analysis of PEDV positive cells in the nasal cavity . All data shown are the mean results ± SD from three independent experiments. Statistical significance was tested using one-way ANOVA. ** P < 0.01.

Article Snippet: The anti-PEDV N protein mAb was purchased from Medgene labs. Anti-pig epithelial cell marker PE- Keratin 18 (CK18) mAb was purchased from Novus Biologicals.

Techniques: Isolation, Infection, Staining